Cloned fusion product from a rare t(15;19)(q13.2;p13.1) inhibit S phase in vitro.

Abstract

Somatically acquired chromosomal translocation is a common mechanism of oncogene activation in many haematopoietic tumours and sarcomas. However, very few recurrent chromosomal translocations have been reported in more common epithelial tumours such as lung carcinomas.